An amazing medical discovery for easy weight loss

For the first time, scientists have been able to find a way to enable people who suffer from obesity and a higher than normal amount of fat to lose weight.

Scientists from the University of California have revealed that by suppressing just one protein, white fat stores can be converted into calorie-burning fat, according to what was published by the New Atlas website.

The researchers found that by suppressing the protein KLF-15, which has previously been identified as having a role in cell metabolism, and which is more abundant in brown and sand-colored calorie-burning fat, the cells themselves change their function.

It also turned out that without the presence of the KLF-15 protein, the default setting for fat cells appears to be beige, not the dominant white.

The findings suggest that suppressing KLF-15 converts white fat cells into brown fat cells, and that the barrier to doing so is not as high as previously thought, according to the journal Clinical Investigation.

Previously, scientists have focused on converting stem cells into brown fat, or brown fat, which burns calories, which the body primarily uses as fuel to regulate temperature.

As such, these cells burn fat stores, while white adipose tissue is the stores that the body has the most difficulty converting during weight loss.

Until now, it was thought that stem cells were required from the start, so by suppressing KLF-15, it could be much easier to accelerate effective weight loss in the long term than previously thought.

Thanks to the vast amount of research in this area, it is known that mammals have a mixture of brown and white fat, with the recently discovered sand-colored adipose tissue lying somewhere in between.

White fat cells are reserve energy stores, which can release fatty acids when energy is needed — but many people can have such an abundant supply that the body can’t easily use them to burn calories and lose weight.

Sandy fat cells burn energy efficiently, like brown fat tissue, but they are spread throughout white fat tissue. So, in experiments with lab mice, turning off KLF-15 caused sandy fat cells to grow in size, sacrificing white fat cells, essentially resulting in a significant reduction in fat stores.

The researchers then transplanted human fat cells to see how KLF-15 could change stores into calorie-burning powerhouses. They discovered that the protein directly controlled a receptor known as Adrb1, opening the door to the possibility of a drug that targets it, and could be a better option for weight loss than GLP-1 treatments with their wide-ranging side effects.